They say you should eat less salt if you have high blood pressure.
What is the relationship between salt and high blood pressure?
676 2016-06-25 Gentleman
Salt intake and high blood pressure are closely related. High blood pressure worsens when salt intake increases, and alleviates when salt intake decreases. Therefore, the habit of eating salty food on a daily basis must be corrected. In addition, the kidneys play a role in cleaning blood by filtering waste from the body, and high blood pressure can occur even when blood is not filtered well due to abnormalities in the kidneys.
[Naver Knowledge Encyclopedia] Hypertension is not a dangerous disease if the cause is found and treated. (Jaseng Oriental Medicine Hospital Health Column, Jaseng Oriental Medicine Hospital)
http://terms.naver.com/entry.nhn?docId=3346974&...
Salt (sodium) affects high blood pressure because an increase in sodium in the body stimulates the nerve nucleus of the hypothalamus and causes abnormal secretion of blood pressure control hormones in the hippocampus, which prevents blood pressure control.
Also, if there is a lot of sodium in the cell, it causes edema and raises blood pressure.
1)a mechanical basis
Uncivilized primitive people who live in remote isolated areas of the earth have no high blood pressure, and with all civilized populations, the moon does not rise in blood pressure as it ages. For example, Yanomamo Indians in northern Brazil excrete only 23 mg of sodium per day (calculated as .58 mg in salt), with an average blood pressure of 107/67 mmHg for men in their 40s and 98/62 mmHg for women. The absence of high blood pressure may be explained by other differences in lifestyle, but comparisons between groups under similar conditions show that blood pressure is most directly related to the level of sodium intake. Moreover, it can be seen that blood pressure rises and high blood pressure occurs as even the uncivilized population without high blood pressure accepts a modern lifestyle that consumes a lot of sodium. In most large-scale population studies, it was found that there was a significant correlation between the level of salt intake, the height of blood pressure, and the frequency of hypertension. Electrolytes and blood pressure were measured 24 hours a day in 579 men and women in their 20s to 50s in 52 locations around the world, with significant correlations between sodium excretion, systolic blood pressure and diastolic blood pressure, especially with age. There was no high blood pressure in the population who consumed an average of less than 1,150 mg of sodium (2900 mg or 2.9 grams of salt) per day, and there was no tendency for blood pressure to rise gradually with age. For example, Alaska Eskimos have a daily salt intake of about 4 grams, which is close to 2 to 3 grams per person's daily requirement, or natural food content, and their hypertension rate is almost zero. On the other hand, it accounts for 11 percent of Americans with 10 grams per day, and 30 percent of Americans with 33 grams per day in northeastern Japan. The salt intake of Koreans is estimated to reach 15 to 20 grams a day, and the frequency of high blood pressure is estimated to be 15 to 20%.
2)an experimental basis
There are many experimental evidence showing that excessive salt intake is associated with high blood pressure. Limiting sodium in hypertensive patients lowers their blood pressure. Strictly restricting sodium may significantly lower blood pressure, but in most studies, blood pressure drops modestly, even if less strictly restricted to 1.7 to 2.3 grams of sodium (4.4 to 5.8 grams of salt) per day. Although it has been found that increasing salt intake in a short period of time increases blood pressure in normal blood pressure, it may not be possible to conclude that increasing salt intake in humans results in high blood pressure. However, in animals that are genetically predisposed to high blood pressure, it is rather easy to do so. Although it was not appropriate to study, starting with infants and young children, preventing high blood pressure by limiting sodium for a long time, or consuming excessive sodium, nearly 500 newborns were divided into two groups, feeding them about half of their sodium intake for a short period of six months and half of their sodium intake after six months. 다른 Blood pressure contracted 2.1mm. Moreover, when hundreds of people with high normal blood pressure were randomly tested with metabolism, the blood pressure was lower in the group that limited sodium intake for 18 months and 5 years than in the group that did not limit sodium intake. A high intake of sodium may activate a number of boost mechanisms. Abnormal vasoconstriction occurs within the renal circulation, and boost reactions to stimulating substances are promoted.
3)sensitivity to salt
There is still controversy among scholars as to whether excessive salt intake is the cause of primary hypertension (essential hypertension), but the above evidence shows that excessive salt intake is closely related to hypertension. So, how can we explain that almost all people in civilized areas eat a high sodium diet, and that only about half of them develop high blood pressure? This difference is explained by the various sensitivity of blood pressure to sodium. In one study, according to the criteria they set, 51% of hypertensive patients were highly susceptible to sodium, compared with only 26% in normal people. In addition, as they get older, their sensitivity to sodium increases in normal people and high blood pressure patients. It has been reported that increased blood pressure also increases the excretion of sodium, whereas those with high sensitivity to sodium have poor control. This sensitivity to sodium tends to inherit, and there are reports that blood pressure changes well when limiting sodium in maternal offspring. It would be reasonable to actively encourage low-salt diets to highly sensitive people if they could distinguish those who are highly sensitive to sodium from those who are not and are highly resistant. Studies on this usually distinguished them according to changes in blood pressure or substances related to blood pressure by low-salt or by excreting salt using diuretics and loading salt. However, these methods have their own problems, so they are not yet readily available in clinical practice, and predictions of sodium sensitivity are not reliable. So for now, we have no choice but to encourage everyone to eat less salt.
Source: http://www.vitamincall.com/2012/05/blog-post_3081...
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